Medin Co-aggregates with Vascular Amyloid-β in Alzheimer’s Disease

Researchers demonstrated in amyloid-β precursor protein transgenic mice and in patients with Alzheimer’s disease that medin co-localized with vascular amyloid-β deposits, and that in mice, medin deficiency reduced vascular amyloid-β deposition by half.
[Nature]
Full Article
spot_img

Stay up-to-date with your field!

Subscribe for free weekly science newsletters.

Related News