GATA6 Triggers Fibroblast Activation and Tracheal Fibrosis through the Wnt/β-Catenin Pathway

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Researchers investigated the potential role of GATA6 as a novel regulator of tracheal fibrosis. It was found that GATA6 and α-smooth muscle actin (α-SMA) were obviously increased in tracheal fibrotic granulations and in TGFβ1-treated primary tracheal fibroblasts.
[Cellular Signalling]
AbstractGraphical Abstract