N-Acetyl-L-Cysteine Attenuates Oxidative Stress-Induced Bone Marrow Endothelial Cells Apoptosis by Inhibiting BAX/Caspase 3 Pathway

Researchers explored injury caused by oxidative stress enhancement in bone marrow endothelial cells (BMECs) both in vivo and in vitro. The BMECs, indicators of oxidative stress, bone mass, and apoptosis-related proteins were analyzed in different age groups.