MAF Amplification Licenses ERα through Epigenetic Remodeling to Drive Breast Cancer Metastasis

The authors integrated proteomics, transcriptomics, epigenomics, chromatin accessibility and functional assays from human and syngeneic mouse breast cancer models to show that MAF directly interacts with estrogen receptor alpha.
[Nature Cell Biology]
Full ArticlePress Release

Stay up-to-date with your field!

Subscribe for free weekly science newsletters.

Related News