SNX16 Is Required for Hepatocellular Carcinoma Survival via Modulating the EGFR-AKT Signaling Pathway

Researchers delved into SNX16’s regulation of the EGFR-AKT signaling cascade mechanism. SNX16 overexpression in HCC correlated with poor patient survival; enhancing proliferation, migration, invasion, and tumorigenicity, while SNX16 knockdown suppressed these processes.