ACSL4-Mediated H3K9 and H3K27 Hyperacetylation Upregulates SNAIL to Drive TNBC Metastasis

By
Emily Salmini
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Loss of acyl-CoA synthetase 4 (ACSL4) function, by genetic ablation or pharmacological inhibition significantly reduced metastatic potential of TNBC.
[Proceedings of The National Academy of Sciences of The United States of America]
Abstract