Inhibition of ICAM1 Diminishes Stemness and Enhances Antitumor Immunity in Glioblastoma via β-catenin/PD-L1 Signaling

The authors identified intercellular adhesion molecule 1 (ICAM1) as a key regulator of glioblastoma stem cell stemness and tumorigenicity, promoting an immunosuppressive microenvironment via β-catenin/PD-L1 signaling.