Synergistic Regulation of TGF-β1/Smad2/3 Signaling and Inflammatory Pathways via SA/NAC-Based Nanoplatforms: A Novel Strategy to Enhance Anti-Fibrotic Therapeutic Outcomes in Idiopathic Pulmonary Fibrosis

The authors demonstrated that macrophage-derived apoptotic body delivery system co-loaded with sodium arsenite (SA) and N-acetylcysteine (NAC) represents an effective therapeutic strategy for idiopathic pulmonary fibrosis.