Fndc5 Modification Optimizes the Therapeutic Effect of Rat MSCs on Sepsis-Induced ALI/ARDS via Activating the PI3K/AKT Signaling Pathway

Fibronectin type III domain-containing protein 5 (Fndc5) modification significantly increased the retention rate of MSCs in sepsis-induced acute lung injury murine model while augmenting their in vitro proliferation and migration potential.