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acute lung injury

Human Placental Hematopoietic Stem Cell Derived Natural Killer Cells (CYNK-001) Mediate Protection against Influenza a Viral Infection

[Human Vaccines & Immunotherapeutics] Scientists investigated the effects of CYNK-001, human placental hematopoietic stem cell derived NK cells that exhibited strong cytolytic activity against a range of malignant cells and expressed high levels of activating receptors, against influenza A virus infections.

Mesenchymal Stem Cell-Derived Extracellular Vesicles in the Management of COVID19-Associated Lung Injury: A Review on Publications, Clinical Trials and Patent Landscape

[Journal of Tissue Engineering and Regenerative Medicine] The authors present the current landscape of the field and highlights some interesting findings on MSC-derived extracellular vesicles in the context of COVID-19, including in silico, in vitro, in vivo and case reports.

Lipidomic Profiling of Bronchoalveolar Lavage Fluid Extracellular Vesicles Indicates Their Involvement in Lipopolysaccharide-Induced Acute Lung Injury

[Journal of innate Immunity] Extracellular vesicles released during acute lung injury originated from alveolar epithelial cells, macrophages, and neutrophils and carry a diverse array of lipid mediators derived from ω-3 and ω-6 polyunsaturated fatty acids.

Inhibition of the C3a Receptor Attenuates Sepsis-Induced Acute Lung Injury by Suppressing Pyroptosis of the Pulmonary Vascular Endothelial Cells

[Free Radical Biology and Medicine] Scientists investigated the important role and mechanism of C3a in preventing the occurrence of pyroptosis to protect the lung endothelial cells in sepsis-induced acute lung injury.

Inhibition of Lung Microbiota-Derived Proapoptotic Peptides Ameliorates Acute Exacerbation of Pulmonary Fibrosis

[Nature Communications] Researchers demonstrated that an anticorisin monoclonal antibody ameliorated lung fibrosis by significantly inhibiting acute exacerbation in the human transforming growth factorβ1 model and acute lung injury in the bleomycin model.

The Suppressive Effects of Mer Inhibition on Inflammatory Responses in the Pathogenesis of LPS-Induced ALI/ARDS

[Science Signaling] In human pulmonary aortic endothelial cells,lipopolysaccharide (LPS) induced decreased in the amounts of endothelial nitric oxide synthase, thrombomodulin, and vascular endothelial–cadherin, which was blocked by treatment with UNC2250.

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