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cell proliferation

Group 1 ILCs Regulate T Cell–Mediated Liver Immunopathology by Controlling Local IL-2 Availability

[Science Immunology] Scientists used multiphoton intravital microscopy in mouse models of hepatitis B to study the intrahepatic behavior of group 1 innate lymphoid cells (ILCs) and their cross-talk with hepatitis B virus–specific CD8+ T cells.

Influence of Trichomonas vaginalis Macrophage Migration Inhibitory Factor on the Proliferation Activity of Prostate Epithelial Cell Line and Its Preliminary Mechanism

[andrology] The authors determined the influence of macrophage Trichomonas vaginalis macrophage migration inhibitory factor on prostate epithelial cells and its preliminary mechanism.

Combination Treatment of Docetaxel with Caffeic Acid Phenethyl Ester Suppresses the Survival and the Proliferation of Docetaxel-Resistant Prostate Cancer Cells via Induction of Apoptosis...

[Journal of Biomedical Science] Combination treatment of docetaxel with caffeic acid phenethyl ester effectively suppressed the proliferation and survival of docetaxel-resistant prostate cancer cells via inhibition of Bcl-2 and c-Myc as well as induction of metabolism interference.

HepaCAM-PIK3CA Axis Regulates the Reprogramming of Glutamine Metabolism to Inhibit Prostate Cancer Cell Proliferation

[International Journal of Oncology] Liquid chromatography‑tandem mass spectrometry and gas chromatography‑mass spectrometry assays were performed, and the results revealed markedly reduced levels of glutamine and metabolic flux in the blood samples of patients with prostate cancer (PCa) and in PCa cells.

Circular RNA-UBE2D2 Accelerates the Proliferation and Metastasis of Non-small Cell Lung Cancer Cells via Modulating microRNA-376a-3p/Eukaryotic Translation Initiation Factor 4γ2 Axis

[Bioengineered] Scientists suggested that circ-UBE2D2 promoted the proliferation, migration and invasion but restrained apoptosis of lung cancer cells by regulating miR-376a-3p/eukaryotic translation initiation factor 4γ2 axis.

Long Noncoding RNA LINC01234 Promotes Hepatocellular Carcinoma Progression through Orchestrating Aspartate Metabolic Reprogramming

[Molecular Therapy] Investigators screened LINC01234 as an aspartate metabolism-related lncRNA in HCC. Clinically, LINC01234 was highly expressed in HCC, and high LINC01234 expression level was correlated with poor prognosis of HCC patients.

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