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endothelial cells

Pseudogene RPL32P3 Regulates the Blood–Tumor Barrier Permeability via the YBX2/HNF4G Axis

[Cell Death Discovery] Researchers identified that ribosomal protein L32 pseudogene 3 (RPL32P3) was highly expressed in glioma-exposed endothelial cells. Knockdown of RPL32P3 decreased the expression of tight junction-related proteins and increased blood–tumor barrier permeability.

Staphylococcus aureus Iron-Regulated Surface Determinant B (IsdB) Protein Interacts with Von Willebrand Factor and Promotes Adherence to Endothelial Cells

[Scientific Reports] The binding of IsdB and adhesion of Staphylococcus aureus expressing IsdB to monolayers of activated endothelial cells was significantly inhibited by a monoclonal antibody against the A1 domain and by IsdB reactive IgG from patients with staphylococcal endocarditis.

Receptor Interacting Protein 3 Kinase, Not 1 Kinase, through MLKL-Mediated Necroptosis Is Involved in UVA-Induced Corneal Endothelium Cell Death

[Cell Death Discovery] The authors investigated receptor-interacting protein 3 kinase, a key signaling molecule of programmed cell death, in UVA-induced injury using a short-term corneal endothelium culture model.

Insulin-Stimulated Adipocytes Secrete Lactate to Promote Endothelial Fatty Acid Uptake and Transport

[Journal of Cell Science] Investigators modeled the relationship of adipocytes and endothelial cells in vitro to test the role of insulin in fatty acid transport.

A 3D Primary Human Cell-Based In Vitro Model of Non-Alcoholic Steatohepatitis for Efficacy Testing of Clinical Drug Candidates

[Scientific Reports] Investigators presented a 3D in vitro microtissue model that used spheroidal, scaffold free co-culture of primary human hepatocytes, Kupffer cells, liver endothelial cells and hepatic stellate cells.

Sevoflurane Preconditioning Promotes Mesenchymal Stem Cells to Relieve Myocardial Ischemia/Reperfusion Injury via TRPC6-Induced Angiogenesis

[Stem Cell Research & Therapy] The authors investigated whether sevoflurane preconditioning could promote MSCs to attenuate myocardial I/R injury via transient receptor potential canonical channel 6 (TRPC6)-induced angiogenesis.

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