endothelial cells

[Stem Cell Research & Therapy] In a wound healing mouse model, the histological evaluation of wound tissue in PBS, bone marrow MSCs (BMSCs), and ascorbic acid 2-glucoside-treated BMSCs group were investigated.

[Nature Communications] Pannexin-1 on endothelial cells acted as a conduit for ATP release that stimulated macrophage activation via P2X7 receptors and mitochondrial DNA release to increase IL-1β and HMGB1 secretion.

[Journal of Nanobiotechnology] VEGF/nerve growth factor (NGF)-nanoparticles (NPs) promoted VEGF and NGF co-expression simultaneously, eliminated excessive H2O2, strengthened reactions between SH-SY5Ys and HUVECs, and finally enhanced migration, tube formation, proliferation and H2O2 damage resistance of HUVECs.

[ACS Nano] Scientists fabricated a micro- to nanoscale network assembly consisting of copper ion and epigallocatechin gallate via π–π interactions, metal coordination, and oxidative polymerization.

[Molecular Therapy-Nucleic Acids] Using a streptozotocin-induced diabetic wound healing mouse model, investigators showed that, compared with miR-scrambled-modulated small extracellular vesicles (sEVs), topical administration of miR-425-5p-modulated sEVs significantly enhanced wound healing, a process mediated by enhanced vascularization and skin re-epithelialization.

[Scientific Reports] Scientists evaluated the impact of morphine exposure at three different concentrations for 24 hours and 48 hours on primary human brain microvascular endothelial cells.