Tag results:
lung cancer cells
Prostate Cell News
Cancer Cells with Defective RB and CDKN2A Are Resistant to the Apoptotic Effects of Rapamycin
[Cancer Letters] Scientists observed that DU145 prostate and NCI–H2228 lung cancer cells were resistant to the cytotoxic effect of rapamycin.
Pulmonary Cell News
Autophagic Secretion of HMGB1 from Cancer-Associated Fibroblasts Promotes Metastatic Potential of Non-Small Cell Lung Cancer Cells via NFκB Signaling
[Cell Death & Disease] Researchers reported that autophagy was upregulated in lung cancer-associated cancer-associated fibroblasts (CAFs) compared to normal fibroblasts, and autophagy was responsible for the promoting effect of CAFs on non-small cell lung cancer cell migration and invasion.
Pulmonary Cell News
MRTF-a-NF-κB/p65 Axis-Mediated PDL1 Transcription and Expression Contributes to Immune Evasion of Non-Small-Cell Lung Cancer via TGF-β
[Experimental and Molecular Medicine] Researchers defined a new signaling pathway that regulated the transcription and expression of PD-L1 upon TGF-β treatment, which might have a significant impact on research into the application of immunotherapy in treating lung cancer.
Pulmonary Cell News
The Petroleum Ether Extract of Brassica rapa L. Induces Apoptosis of Lung Adenocarcinoma Cells via the Mitochondria-Dependent Pathway
[Food & Function] The effects and pharmacological mechanisms of BRPS on the treatment of lung cancer were investigated both in vitro and in vivo.
Pulmonary Cell News
Propyl Gallate Decreases the Proliferation of Calu-6 and A549 Lung Cancer Cells via Affecting Reactive Oxygen Species and Glutathione Levels
[Journal of Applied Toxicology] Calu-6 and A549 lung cancer cells were used to examine the anti-proliferative effect of propyl gallate in relation to reactive oxygen species and glutathione levels.
Prostate Cell News
Methylseleninic Acid Overcomes Programmed Death-Ligand 1-Mediated Resistance of Prostate Cancer and Lung Cancer
[Molecular Carcinogenesis] Scientists showed that methylseleninic acid substantially attenuated cisplatin-induced PD-L1 expression via inhibiting protein kinase B phosphorylation, thereby potentiated cisplatin cytotoxicity in prostate and lung cancer cell models.