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qRT-PCR

Ascorbic Acid 2-Glucoside Preconditioning Enhances the Ability of Bone Marrow Mesenchymal Stem Cells in Promoting Wound Healing

[Stem Cell Research & Therapy] In a wound healing mouse model, the histological evaluation of wound tissue in PBS, bone marrow MSCs (BMSCs), and ascorbic acid 2-glucoside-treated BMSCs group were investigated.

MicroRNA-98-5p Inhibits IL-13-Induced Proliferation and Migration of Human Airway Smooth Muscle Cells by Targeting RAC1

[Inflammation] Investigators focused on how miR-98-5p functioned in the proliferation and migration of human airway smooth muscle cells treated with interleukin-13 (IL-13).

Catalpol Protects AC16 Cells from Hypoxia/Reoxygenation Injury by Regulating the miR-22-3p/DPP4 Axis

[Journal of Biochemical and Molecular Toxicology] Catalpol promoted the viability and reduced cell apoptosis of human cardiomyocyte AC16 cells and repressed the release of inflammatory cytokines TNF-α, IL-6, and IL-1β, while inhibiting the leakage of myocardial injury markers lactate dehydrogenase and creatine kinase.

OSI-027 Inhibits the Tumorigenesis of Colon Cancer Through Mediation of C-myc/FOXO3a/PUMA Axis

[Cell Biology International] OSI-027 dose-dependently reduced colon cancer cell viability through inducing the cell apoptosis and induced the apoptosis of colon cancer cells via upregulation of p53-upregulated modulator of apoptosis (PUMA).

Golgi Phosphoprotein 3 Promotes Colon Cancer Cell Metastasis Through STAT3 and Integrin α3 Pathways

[Frontiers in Molecular Biosciences] In vitro genetic ablation of golgi phosphoprotein 3 (GOLPH3) was performed using small interfering RNA transfection, and a stably overexpressed GOLPH3 colon cancer cell line was constructed using the lentivirus system.

Extracellular Vesicle-Mediated Delivery of miR-127-3p Inhibits the Proliferation and Invasion of Choriocarcinoma Cells by Targeting ITGA6

[Experimental Cell Research] Scientists studied the effects of miR-127-3p-enriched extracellular vesicles on choriocarcinoma and underlying mechanisms.

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