USP28 Enables Oncogenic Transformation of Respiratory Cells and Its Inhibition Potentiates Molecular Therapy Targeting Mutant EGFR, BRAF and PI3K.

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Targeting ubiquitin carboxyl-terminal hydrolase 28 (USP28) protein abundance at an early stage via inhibition of its activity is a feasible strategy for the treatment of early-stage lung tumors.
[Molecular Oncology]
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