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The Suppressive Effects of Mer Inhibition on Inflammatory Responses in the Pathogenesis of LPS-Induced ALI/ARDS

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In human pulmonary aortic endothelial cells,lipopolysaccharide (LPS) induced decreased in the amounts of endothelial nitric oxide synthase, thrombomodulin, and vascular endothelial–cadherin, which was blocked by treatment with UNC2250.
[Science Signaling]

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Abstract
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