Scientists showed that the N-linked glycosylation at residue 105 in the first immunoglobulin-like domain of CD155 was involved in the binding of CD155 to both DNAM-1 and tyrosine-based inhibitory motif domain.
[International Immunology]
Essential Role of CD155 Glycosylation in Functional Binding to DNAM-1 on Natural Killer Cells
