Researchers investigated the protective effect of emodin on septic myocardial injury and the underlying mechanism. Lipopolysaccharide (LPS)-induced C57BL/6 mice and cardiomyocytes were used as models of sepsis in vivo and in vitro, respectively.
[Phytotherapy Research]
7992332
WSSMVGPX
items
1
apa
0
default
asc
1
163664
https://www.stemcellsciencenews.com/wp-content/plugins/zotpress/