Investigators developed a mouse model in which a mutated form of HIF1α could be inducibly expressed in cardiomyocytes, and suggested that loss of calcium/calmodulin dependent protein kinase II, gamma (CAMK2G) after myocardial infarction was HIF1-dependent and may have played an important role in the heart’s calcium signaling and response to hypoxia.
[Scientific Reports]
7992332
7SQSHJRI
items
1
apa
0
default
asc
1
163872
https://www.stemcellsciencenews.com/wp-content/plugins/zotpress/
