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KCNN4-Mediated Ca2+/MET/AKT Axis Is Promising for Targeted Therapy of Pancreatic Ductal Adenocarcinoma

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Researchers demonstrated that the level of KCNN4 in pancreatic ductal adenocarcinoma was abnormally elevated, and the overexpression of KCNN4 was induced by transcription factor AP-1.
[Acta Pharmacologica Sinica]
7753456 {7753456:WZMSAPJ6} apa 50 1 164005 https://www.stemcellsciencenews.com/wp-content/plugins/zotpress/
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