The authors indicated that CMDL-1 may have played an anti-apoptotic role in doxorubicin (DOX) cardiotoxicity by regulating Drp1 S637 phosphorylation. Thus, CMDL-1 may have served as a potential therapeutic target in DOX cardiotoxicity.
[Molecular Therapy-Nucleic Acids]
7992332
{7992332:L95T6ALW}
apa
50
1
165974
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