Home Newsletters Intestinal Cell News Intestinal Gpr17 Deficiency Improves Glucose Metabolism by Promoting GLP-1 Secretion

Intestinal Gpr17 Deficiency Improves Glucose Metabolism by Promoting GLP-1 Secretion

0
Scientists showed that Gpr17, an orphan G protein-coupled receptors, was co-expressed in glucagon-like peptide-1 (GLP-1)-expressing EECs in human and rodent intestinal epithelium.
[Cell Reports]

Sorry, but the selected Zotpress account can't be found.

Full ArticleGraphical Abstract
Exit mobile version