Antiphospholipid autoantibodies increased human endometrial stromal cell decidualization, senescence and inflammation. The decidualization and inflammatory responses were partially mediated by toll-like receptor 4 (TLR4) and p38 MAP kinase, while the decidualization and senescence responses were reactive oxygen species (ROS)-dependent.
[Arthritis & Rheumatology]
7992332
{7992332:BBBBBBBB}
apa
50
1
171079
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