ACLS4 Could Be a Potential Therapeutic Target for Severe Acute Pancreatitis

The authors conducted a comprehensive transcriptomic analysis of peripheral blood from patients with acute pancreatitis (AP) and the pancreatic tissue from a mouse model of AP. In mouse models of severe AP, ACSL4 was significantly upregulated in the pancreas, whereas GALNT3, WSB1, and IL1R1 were not.