 | | Vol. 12.21 – 01 Jun, 2023 |
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| Scientists used transcriptome sequencing to determine the transcriptional response of the A549 type II alveolar epithelial cell line to infection with strains CEA10 and Af293, two clinical isolates of A. fumigatus.
[Microbiology Spectrum] |
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 | | PUBLICATIONSRanked by the impact factor of the journal |
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| Researchers showed that extracellular vesicles secreted by primary human alveolar epithelial cells transfered multiple distinct sRNA fragments to P. aeruginosa that were predicted to target the three subunits of the fluoroquinolone efflux pump MexHI-OpmD, thus increasing antibiotic sensitivity.
[American Journal Of Physiology-Lung Cellular And Molecular Physiology] |
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| Investigators examined the capacity of alveolar epithelial cells to produce extracellular matrix and thereby directly contribute towards remodeling in chronic lung diseases.
[Scientific Reports] |
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| Scientists determined D-cycloserine to be the optimal candidate for anti-tuberculosis cell therapy due to its effectiveness, relatively short biosynthetic pathway, and its low-resistance incidence.
[Scientific Reports] |
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| The prevention and treatment effects of fibroblast growth factor-21 in bleomycin (BLM)-induced pulmonary fibrosis was investigated in vivo and in vitro.
[Naunyn-Schmiedebergs Archives Of Pharmacology] |
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| SRY-box transcription factor 9 (SOX9) consistently drove organoid growth in vitro, but SOX9-promoted tumor growth was significantly attenuated in immunocompromised mice compared to syngeneic mice.
[Oncogene] |
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| Researchers screened for chemosensitivity-regulating tripartite motif (TRIM) proteins using cisplatin (CDDP)-resistant NSCLC cell lines. They showed that TRIM17 was upregulated in CDDP-resistant NSCLC cells and tumors compared to CDDP-sensitive counterparts.
[Cellular Oncology] |
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| Investigators reported that KLF transcription factor 11 (KLF11) inhibited lung adenocarcinoma cell proliferation and promoted chemotherapy sensitivity by participating in the glutathione peroxidase 4 (GPX4)-related ferroptosis pathway.
[Communications Biology] |
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| To investigate the role of SLITRK6 in lung adenocarcinoma (LUAD) and the underlying mechanisms, clinical tissues, and tissue microarray of LUAD were used to detect the expression of SLITRK6.
[Apoptosis] |
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| The authors discuss potential mechanisms of transformation and review the current knowledge about cells of origin of NSCLC and SCLC. They also discuss treatment options for transformed SCLC, including chemotherapy, radiotherapy, tyrosine kinase inhibitors, immunotherapy, and anti-angiogenic agents.
[Seminars In Cancer Biology] |
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| C4 Therapeutics, Inc. announced an exclusive licensing agreement for the development and commercialization of CFT8919 in Greater China , including Hong Kong SAR, Macau SAR, and Taiwan.
[C4 Therapeutics, Inc.] |
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| June 25 – 30, 2023
Newry, Maine, United States |
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| | Institut Pasteur – Paris, France |
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| | Rutgers University – New Brunswick, New Jersey, United States |
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| | University of Washington – Medical Lake, Washington, United States |
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| | St. Jude Children’s Research Hospital – Memphis, Tennessee, United States |
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| | National Insitute of Health – Bethesda, Maryland, United States |
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