Elevated circASCC3 Limits Antitumor Immunity by Sponging miR-432–5p To Upregulate C5a in Non-Small Cell Lung Cancer

The expression of circASCC3 in NSCLC tissues and cell lines was evaluated by fluorescence in situ hybridization and quantitative reverse transcription-polymerase chain reaction. The functions and mechanisms of circASCC3 in NSCLC progression and anti-programmed cell death 1 resistance were uncovered in vitro and in vivo.