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Pulmonary Cell NewsInvestigators reported that overexpression of Schlafen-5 (SLFN5) inhibited HIV-1 replication and reduced viral mRNA levels, whereas depletion of endogenous SLFN5 promoted HIV-1 replication.
[Nucleic Acids Research]
Home Newsletters Immunology of Infectious Disease News Schlafen 5 Suppresses Human Immunodeficiency Virus Type 1 Transcription by Commandeering Cellular...
