AKT-Driven Epithelial-Mesenchymal Transition Is Affected by Copper Bioavailability in HER2 Negative Breast Cancer Cells via a LOXL2-Independent Mechanism

Scientists exposed different breast cancer cell lines, including two TNBC ones, an human epidermal receptor 2 (HER2) enriched, and one cell line representative of the Luminal A molecular subtype, to short- or long-term copper-chelation by triethylenetetramine.