Using Sendai virus to model natural infection in mice, investigators found evidence that alveolar type 1 cells had an intermediary role by persisting in areas depleted of alveolar type 2 cells, upregulating interferon responsive genes, and receding from invading airway cells.
[American Journal of Respiratory Cell and Molecular Biology]

Cancer Stem Cell News
Cell Therapy News
Dermal Cell News
Endothelial Cell News
ESC & iPSC News
Extracellular Matrix News
Hematopoiesis News
Hepatic Cell News
Human Immunology News
Immune Regulation News
Intestinal Cell News
Mammary Cell News
Mesenchymal Cell News
Muscle Cell News
Neural Cell News
Organoid News
Pancreatic Cell News
Prostate Cell News
Pulmonary Cell News