Expression of DEL-1 in Alveolar Epithelial Cells Prevents Lipopolysaccharide-Induced Inflammation, Oxidative Stress, and Eosinophil Recruitment in Acute Lung Injury

Researchers investigated the role of developmental endothelial locus-1 (DEL-1) in lipopolysaccharide-induced acute lung injury in mouse models and its ability to regulate on eosinophil recruitment.