ABRO1 Arrests Postnatal Cardiomyocyte Proliferation and Myocardial Repair By Blocking N6-Methyladenosine Methylation and Expression of PSPH

Scientists revealed that abraxas 2 (ABRO1) is an imperative contributor to the cell cycle withdrawal and attenuation of proliferative response in the postnatal cardiomyocytes and could act as a potential target to accelerate cardiomyocyte proliferation and cardiac repair in the adult heart.