Transient Receptor Vanilloid Subtype 4 (TRPV4)-Mediated Ca2+ Influx Promotes Glomerular Endothelial Inflammation in Sepsis-Associated Acute Kidney Injury

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Investigators found that TRPV4 expression in mouse glomerular endothelial cells (MGECs) increased after lipopolysaccharide stimulation or cecal ligation and puncture challenge, which increased intracellular Ca2+ in MGECs.
[Laboratory Investigation]
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