Triiodothyronine Positively Regulates Endoplasmic Reticulum-Associated Degradation (ERAD) and Promotes Androgenic Signaling in Androgen-Dependent Prostate Cancer Cells

Scientists investigated the molecular action of triiodothyronine (T3) on the ERAD mechanism and unfolded protein response signaling in prostate cancer cells and also extensively examined the effect of T3 on androgenic signaling.