TNFRSF19 Promotes Endoplasmic Reticulum Stress-Induced Paraptosis via the Activation of the MAPK Pathway in Triple-Negative Breast Cancer Cells

Scientists demonstrated that the upregulation of TNFRSF19 functioned as a tumor suppressor in TNBC by stimulating paraptosis through the activation of the MAPK pathway-mediated endoplasmic reticulum stress.