Reprogramming of Palmitic Acid Induced by Dephosphorylation of ACOX1 Promotes β-Catenin Palmitoylation To Drive Colorectal Cancer Progression

Functionally, ACOX1 depletion promoted colorectal cancer cell proliferation in vitro and colorectal tumorigenesis in mouse models, whereas ACOX1 overexpression inhibited patient-derived xenograft growth.