Cardiomyocyte PANX1 Controls Glycolysis and Neutrophil Recruitment in Hypertrophy

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Researchers generated a novel mouse line with constitutive deletion of PANX1 in cardiomyocytes. PANX1 deletion in cardiomyocytes had no effect on unstressed heart function but increased the glycolytic metabolism and resulting glycolytic ATP production.
[Circulation Research]
Abstract