Hyperbaric Oxygen Therapy Alleviates Acute Carbon Monoxide Poisoning-Induced Transition of Cardiac Fibroblast to Myofibroblast and Ameliorates Cardiac Fibrosis via the NRF2/ROS/TGFβ1/Smad Pathway

Hyperbaric oxygen therapy effectively represses acute carbon monoxide poisoning-induced cardiac fibroblast-to-myofibroblast transition and ameliorates cardiac fibrosis by modulating the NRF2/ROS/TGF-β1/Smad signaling pathway.