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cell proliferation

Regulatory Mechanism of Androgen Receptor on NCAPD3 Gene Expression in Prostate Cancer

[Prostate] NCAPD3 is an androgen/AR axis-targeted gene and is involved in androgen receptor (AR)-induced prostate cancer (PCa) cell proliferation, migration, and invasion in vitro and in vivo. Androgen treatment and AR overexpression increased the expression of NCAPD3 in PCa cell lines.

Small Intestinal Submucosa Membrane Modified by Fusion Peptide-Mediated Extracellular Vesicles to Promote Tissue Regeneration

[Advanced Healthcare Materials] Extracellular vesicles (EVs) derived from umbilical cord MSCs can achieve rapid cell proliferation and migration with little immune response by creating a satisfactory microenvironment. Fusion peptide-mediated EVs were able to modify the surface of the small intestinal submucosa membrane via specific combination.

Hsa_circ_0011385 Knockdown Represses Cell Proliferation in Hepatocellular Carcinoma

[Cell Death Discovery] High expression of circ_0011385 was observed in hepatocellular carcinoma tissues and cell lines and was significantly associated with tumor size, TNM stage, and prognosis.

RNF144A-AS1, a TGF-β1- and Hypoxia-Inducible Gene That Promotes Tumor Metastasis and Proliferation via Targeting the miR-30c-2-3p/LOX Axis in Gastric Cancer

[Cell & Bioscience] Scientists explored the function and characterize the underlying mechanism of long noncoding RNA RNF144A-AS1 in gastric cancer metastasis and outgrowth.

The Non-Coding RNA Interactome in Joint Health and Disease

[Nature Reviews Rheumatology] Scientists synthesize literature published in the past two years on the role of non-coding RNAs in osteoarthritis and rheumatoid arthritis with a focus on inflammation, cell death, cell proliferation and ECM dysregulation.

BTN2A2 Protein Negatively Regulates T Cells to Ameliorate Collagen-Induced Arthritis in Mice

[Scientific Reports] Researchers investigated the ability of a recombinant BTN2A2-IgG2a Fc fusion protein to treat Collagen type II (CIA). They showed that administration of BTN2A2-Ig attenuatex established CIA, as compared with control Ig protein treatment.

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