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pulmonary cells

Propyl Gallate Decreases the Proliferation of Calu-6 and A549 Lung Cancer Cells via Affecting Reactive Oxygen Species and Glutathione Levels

[Journal of Applied Toxicology] Calu-6 and A549 lung cancer cells were used to examine the anti-proliferative effect of propyl gallate in relation to reactive oxygen species and glutathione levels.

Electronic Cigarette Smoke Reduces Ribosomal Protein Gene Expression to Impair Protein Synthesis in Primary Human Airway Epithelial Cells

[Scientific Reports] Researchers reported transcriptomic changes, including decreased expression of many ribosomal genes, in airway epithelial cells in response to e-cig exposure.

Anti-inflammatory Actions of Aspirin-Triggered Resolvin D1 (AT-RvD1) in Bronchial Epithelial Cells Infected with Cryptococcus neoformans

[inflammopharmacology] The interaction of Cryptococcus neoformans with airway epithelial cells is crucial for the establishment of cryptococcosis. Scientists evaluated the effects of AT-RvD1 on human bronchial epithelial cells stimulated with C. neoformans.

Methylseleninic Acid Overcomes Programmed Death-Ligand 1-Mediated Resistance of Prostate Cancer and Lung Cancer

[Molecular Carcinogenesis] Scientists showed that methylseleninic acid substantially attenuated cisplatin-induced PD-L1 expression via inhibiting protein kinase B phosphorylation, thereby potentiated cisplatin cytotoxicity in prostate and lung cancer cell models.

Cells of the Human Respiratory Tract Support the Replication of Pathogenic Old World Orthohantavirus Puumala

[Virology Journal] Researchers analyzed the permissiveness of primary endothelial cells of the human pulmonary microvasculature and of primary human epithelial cells derived from bronchi, bronchioles and alveoli for Old World orthohantavirus Puumala virus in vitro.

Signaling Pathway Perturbation Analysis for Assessment of Biological Impact of Cigarette Smoke on Lung Cells

[Scientific Reports] Investigators reported the development of an algorithm based on quantitative assessment of transcriptomic profiles and signaling pathway perturbation analysis of human bronchial epithelial cells exposed to the toxic components present in cigarette smoke.

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