Loss of Endothelial TSPAN12 Promotes Fibrostenotic Eosinophilic Esophagitis via Endothelial Cell–Fibroblast Crosstalk

Scientists proposed that IL-13 decreased TSPAN12, likely contributing to the chronicity of eosinophilic esophagitis by promoting tissue remodeling through fibroblast-endothelial cell crosstalk.