A MYBL2 Complex for RRM2 Transactivation and the Synthetic Effect of MYBL2 Knockdown with WEE1 Inhibition against Colorectal Cancer

Ectopic expression and knockdown experiments indicated that MYBL2 was essential for colorectal cell proliferation, DNA synthesis, and cell cycle progression in an ribonucleotide reductase small subunit M2 (RRM2)-dependent manner.