LINC00337 Induces Tumor Development and Chemoresistance to Paclitaxel of Breast Cancer by Recruiting M2 Tumor-Associated Macrophages

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Investigators focused on the potential involvement and mechanism of LINC00337 in breast cancer (BCa). High-expressed LINC00337 accelerated viability and proliferation of BCa cells, improved the resistance of BCa cells to paclitaxel, and accelerated tumor growth.
[Molecular Immunology]
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Abstract