STIM1-Dependent Peripheral Coupling Governs the Contractility of Vascular Smooth Muscle Cells

The authors found that the number and size of sarcoplasmic reticulum (SR)-plasma membrane interacting sites were decreased, and SR-dependent Ca²⁺ signaling processes were disrupted in freshly isolated cerebral artery SMCs from tamoxifen-inducible, SMC-specific STIM1-knockout mice.