IL-26 Mediates Epidermal Growth Factor Receptor-Tyrosine Kinase Inhibitor Resistance through Endoplasmic Reticulum Stress Signaling Pathway in Triple-Negative Breast Cancer Cells

Investigators provided novel insights into the mechanisms of epidermal growth factor receptor-tyrosine kinase inhibitor resistance in TNBC via interaction of IL-26 with a newly identified receptor, and suggested IL-26 as a possible therapeutic target in TNBC.