METTL3 Improves Cardiomyocyte Proliferation upon Myocardial Infarction via Upregulating miR-17-3p in a DGCR8-Dependent Manner

Due to the role of methyltransferase-like 3 (METTL3) in the physiological proliferation of cardiomyocytes, researchers aimed to determine whether METTL3 could also promote cardiomyocyte proliferation under pathological conditions and to elucidate the underlying mechanism.