Transcriptional Regulation and Ubiquitination-Dependent Regulation of HnRNPK Oncogenic Function in Prostate Tumorigenesis

Researchers noted that heterogeneous nuclear ribonucleoprotein K (HnRNPK) emerged as an important player in the carcinogenesis process of prostate cancer (PrCa). miR-206 and miR-613 suppressed HnRNPK expression by targeting its 3’-UTR in PrCa cell lines in which HnRNPK was overexpressed.