Deficiency of Vitamin D Receptor in Keratinocytes Augments Dermal Fibrosis and Inflammation in a Mouse Model of HOCl-Induced Scleroderma

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The authors demonstrated that the ablation of vitamin D receptor in keratinocytes greatly exacerbated dermal fibrosis in HOCl-induced scleroderma in mice.
[Biochemical and Biophysical Research Communications]

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Abstract