Cardiomyocyte-Specific Deletion of β-Catenin Protects Mouse Hearts from Ventricular Arrhythmias after Myocardial Infarction

The authors demonstrated that entricular tachycardias susceptibility in the chronic phase after myocardial infarction was reduced in mice with cardiomyocyte-specific β-catenin deletion primarily through attenuated structural remodeling, but not ion channel gene alterations.