SLC15A4 Mediates M1-Prone Metabolic Shifts in Macrophages and Guards Immune Cells from Metabolic Stress

By
Justin.choi
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Researchers investigated the mechanism of how SLC15A4 directed inflammatory responses. Proximity-dependent biotin identification revealed glycolysis as highly enriched gene ontology terms.
[Proceedings of the National Academy of Sciences of the United States of America]
Abstract